Fluoride causes Heart Disease, Stroke and Sudden Death
Fluoride causes Heart Disease, Stroke and Sudden Death Geoff Pain February 2016 Abstract Fluoride uptake in coronary arteries is associated with an increased cardiovascular risk of sudden death. Fluoride causes enhanced deposition of doped Hydroxyapatite which leads to inflammation and sites for fatty deposits of atherosclerosis. Fluoride interferes with numerous enzyme systems resulting in elevation of key risk factors for cardiovascular disease.
Fluoride causes oxidative stress and degeneration of heart muscle. Fluoride inhibits Thyroid function with consequential damage to heart function. Fluoride increases risk of sudden death from ruptured aorta. Diabetes, caused or exacerbated by Fluoride, further increases risk of cardiovascular death and disability. Keywords: Fluoride, Fluoridation, Aorta, Apoptosis, Arrhythmia, Atherosclerosis, Bioaccumulation, Calcification, Calcitonin, Cardiovascular, Cerebrovascular, Diabetes, Enzyme Inhibition, Haptoglobin, Heart, Hydroxyapatite, Hyperhomocysteinemia, Hypertension, Inflammation, Interleukin, Lead, Melatonin, Monocyte chemotactic protein-1, Oxidative Stress, Periostin, Plumbosolvency, P-selectin, Review, Ryanodine receptor, Stroke, Thyroid, Vascular Cell Adhesion Molecule-1 Fluoride is a known Heart Poison
The Australian Institute of Health and Welfare states: “Coronary heart disease is the leading underlying cause of death in Australia, followed by dementia and Alzheimer disease, and cerebrovascular diseases (which includes stroke).” [AIHW 2016]. According to the Heart Research Institute “Every 12 minutes, an Australian dies from cardiovascular disease” [HRI 2016]. “Cardiovascular disease is the biggest killer in Australia and worldwide. It was responsible for a third of all Australian deaths in 2010, killing 45,500 people and affecting two out of every three families. In 2009-10, there were 482,000 hospitalisations directly caused by heart disease, and 800,000 hospitalisations where heart disease was a secondary factor.” Fluoride continuously accumulates in the human body over a lifetime [Pain 2015b and references therein] including the heart [Stookey 1963, Panneerselvam 2015] and arteries [Greever 1971, Susheela 1990].
The accumulation of Fluoride involves microcalcification which is discussed in the next section. The toxic impact of Fluoride on the heart and arteries has been studied by hundreds of scientists and carefully reviewed but has not attracted the interest of the media or science journalists, therefore the general public and politicians remain ignorant of the risks [Agalakova 2012, Blank 2012, Waugh 2013, FAN 2016]. The manufacturers of Fluorosilicic Acid used in fluoridation of public drinking water are aware that acute exposure to the toxin is likely to induce ventricular fibrillation and cardiac arrest [CSBP 2005
Fluoride causes oxidative stress and degeneration of heart muscle. Fluoride inhibits Thyroid function with consequential damage to heart function. Fluoride increases risk of sudden death from ruptured aorta. Diabetes, caused or exacerbated by Fluoride, further increases risk of cardiovascular death and disability. Keywords: Fluoride, Fluoridation, Aorta, Apoptosis, Arrhythmia, Atherosclerosis, Bioaccumulation, Calcification, Calcitonin, Cardiovascular, Cerebrovascular, Diabetes, Enzyme Inhibition, Haptoglobin, Heart, Hydroxyapatite, Hyperhomocysteinemia, Hypertension, Inflammation, Interleukin, Lead, Melatonin, Monocyte chemotactic protein-1, Oxidative Stress, Periostin, Plumbosolvency, P-selectin, Review, Ryanodine receptor, Stroke, Thyroid, Vascular Cell Adhesion Molecule-1 Fluoride is a known Heart Poison
The Australian Institute of Health and Welfare states: “Coronary heart disease is the leading underlying cause of death in Australia, followed by dementia and Alzheimer disease, and cerebrovascular diseases (which includes stroke).” [AIHW 2016]. According to the Heart Research Institute “Every 12 minutes, an Australian dies from cardiovascular disease” [HRI 2016]. “Cardiovascular disease is the biggest killer in Australia and worldwide. It was responsible for a third of all Australian deaths in 2010, killing 45,500 people and affecting two out of every three families. In 2009-10, there were 482,000 hospitalisations directly caused by heart disease, and 800,000 hospitalisations where heart disease was a secondary factor.” Fluoride continuously accumulates in the human body over a lifetime [Pain 2015b and references therein] including the heart [Stookey 1963, Panneerselvam 2015] and arteries [Greever 1971, Susheela 1990].
The accumulation of Fluoride involves microcalcification which is discussed in the next section. The toxic impact of Fluoride on the heart and arteries has been studied by hundreds of scientists and carefully reviewed but has not attracted the interest of the media or science journalists, therefore the general public and politicians remain ignorant of the risks [Agalakova 2012, Blank 2012, Waugh 2013, FAN 2016]. The manufacturers of Fluorosilicic Acid used in fluoridation of public drinking water are aware that acute exposure to the toxin is likely to induce ventricular fibrillation and cardiac arrest [CSBP 2005
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